Researchers have recently discovered a single type of mutation in red blood cells that can help the body fight malaria. This discovery can help us understand the mechanism of action of the body against malaria and provide ideas for the development of new therapies.
Recently, researchers from the UK have performed genome-wide sequencing of thousands of primitive tribes in the Sahara region to find a variety of different blood cell markers associated with malaria.
Malaria is a disease caused by five single-cell biological infections from the genus Plasmodium, which can spread through the bite of mosquitoes and can invade the red blood cells of the host for proliferation. Rapid growth and proliferation of red blood cells exacerbate disease symptoms, including high fever, sweating, trembling, pain, and death.
In order to enter the cell, Plasmodium has evolved a number of ways to recognize red blood cell surface receptors. In turn, they can help them invade. In addition, Plasmodium is capable of producing a range of proteins that are linked to receptors on the surface of target cells. These proteins are also the development of current specific vaccines.
So far, we have identified more than a dozen receptor proteins, including A that determine our blood type. B protein. Of course, not all Plasmodium recognize the same receptor. Researchers are also trying to figure out the relationship between different receptors and how they are infected so that we can better prevent and treat malaria.
In a recent study, the authors sequenced and compared the genomes of thousands of people with malaria and normal people. Later, the researchers found that there was a multiplication phenomenon around the blood glycoprotein gene, and there were 27 mutations in the replicated region.
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